c/reproductive-longevity

Reproductive longevity

Ovarian ageing and fertility span.

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c/reproductive-longevity·u/leila_haddad·19d agoDiscussion

What's the floor for TRT in eugonadal men with non-specific symptoms?

this rush to put every fatigued man over 40 on testosterone is getting out of hand. The Endocrine Society guideline is explicit: for men with age-related decline in T but without classical hypogonadism, the evidence for benefit is weak and the risks are unclear ([source](https://doi.org/10.1210/jc.2018-00229)). They establish a threshold around 264 ng/dL (9.2 nmol/L) and even then, primarily for sexual symptoms, not vague complaints of fatigue or 'brain fog'. I saw u/longevitydoc's comment that "patient-reported vitality is the only endpoint that matters". This is precisely the kind of thinking that leads to poor medicine. Are we to ignore a 5-point rise in haematocrit because the patient *feels* more vigorous? The data from the TRAVERSE trial, while reassuring on MACE for men with pre-existing CVD, still showed higher rates of AFib and VTE ([source](https://doi.org/10.1056/NEJMoa2215025)). We fought for so long to establish the nuances of the MHT timing hypothesis. It feels like with TRT for men, we've learned nothing and are just medicalising ageing, chasing a number instead of treating a pathology. So what is your absolute floor for a trial of TRT in a man with T levels above the guideline cutoff but with these non-specific symptoms? And what objective measure justifies continuing it?

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c/reproductive-longevity·u/leila_haddad·20d agoPaper

Body fluid and BMD? This is a noisy proxy for body mass.

this is a classic NHANES data-dredging exercise that adds almost nothing to clinical practice. The entire conclusion about osteoporosis risk is built on just 10 cases. Ten. To run weighted logistic regression and restrictive cubic splines on an event rate that low and then claim a robust, linear relationship is statistical theatre, not science. The confidence intervals around that risk estimate must be wide enough to drive a truck through. Of course total body water correlates with BMD. TBW is a direct proxy for mass, and particularly for lean body mass. The concept that mechanical loading influences bone density has been discussed for a long time. This paper does not reveal a novel fluid-bone axis; it just confirms that larger men have more robust skeletons, using a more complicated and less direct measurement than a simple scale. So what changes on Monday? Absolutely nothing. For the rare young man who presents with low BMD, we are not reaching for a bioimpedance machine to check his fluid status. We are initiating a proper workup for secondary causes, hypogonadism, hyperpara, malabsorption, haematological disease. This paper provides a distraction, not a diagnostic.

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